Jentsch said. But we have found that KCNQ4 is currently not only in ears, but also in some sensory cells in the skin This gave us the idea that the mutation could also affect the sense of touch and that is we we used in our research, closely closely show collaboration with the laboratory of Gary Lewin, a colleague from the MDC, specialized specializes in. . .

The pressure of the sound waves cause thousands of tiny sensitive hairs in the inner ear to vibrate causing the hair cells to fire tiny electric signals . These electrical signals are then travel to the cochlear nerves of the auditory pathway to the brain. The potassium ion flow through a channel in the cell membrane and then from the hair cells. In hearing-impaired persons this potassium channel, a protein molecule as the KCNQ4 – the mutation.A new role for the protein is ABIN-1 produced by our body that usually in small quantities. Add inflammations liver, undue TNF production activates which mechanisms to death death by liver cells and liver fabrics. In addition, excessive TNF supports in the hepatic cells, which the protein LF? responsible for responsible for the inflammation of the liver. This makes it NF? B an attractive target for a therapy would then neutralize the infection. However, an ideal therapy the death of the death of liver cells.